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Tytuł oryginału: Alcohol induced ketoacidosis, severe hypoglycemia and irreversible encephalopathy.
Autorzy: Jain Hitender, Beriwal Sushil, Singh Shailender
Źródło: Med. Sci. Monitor 2002: 8 (11) s.CR77-CR79, il., bibliogr. 4 poz.
Sygnatura GBL: 313,278

Hasła klasyfikacyjne GBL:
  • toksykologia
  • psychiatria i psychologia
  • neurologia

    Typ dokumentu:
  • tytuł obcojęzyczny
  • praca kazuistyczna

    Wskaźnik treści:
  • ludzie
  • dorośli 45-64 r.ż.
  • płeć żeńska

    Streszczenie angielskie: Background: Severe hypoglycemia leading to permanent brain damage is rare in non-daibetic population. We present one such case where chronic alcoholism combined with prologned fasting lead to such a state and patient ended in a persistent vegetative state. Case report: A 50 year old Asian woman with past history of chronic alcoholism and hepatitis C was found unresponsive in her house after drinking alcohol consecutively for 2 days. Finger-stick glucose done by paramedics was 20 mg/dl. She was given 50 ml of 50 p.c. dextrose without any neurological response. On arrival in the emergency department patient was found to be comatose with only withdrawal response to deep painful stimuli and a negative babinski's sign. Computered tomogram of the head revealed mild cerebral atrophy. After an extensive work up a diagnosis of alcoholic ketoacidosis with hypoglycemia related encephalopathy was made. Conclusion: Alcohol induced ketoacidosis is usually associated with normal blood glucose. The probable etiology of low blood sugar in our patient was alcohol-induced inhibition of gluconeogenesis along with starvation. The prolonged hypoglycemia caused cortical damage simulating ischemic brain damage. Ten months in to follow-up patient is still in persistent vegetative state with no noticeable neurological recovery.

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